Why do people start smoking?
Experimenting with smoking usually occurs in the early teenage years
and is driven predominantly by psychosocial motives. For a beginner,
smoking a cigarette is a symbolic act conveying messages such as, in
the words of the tobacco company Philip Morris, “I am no longer my
mother’s child,” and “I am tough.” Children who are attracted to
this adolescent assertion of perceived adulthood or rebelliousness
tend to come from backgrounds that favour smoking (for example, with
high levels of smoking in parents, siblings, and peers; relatively
deprived neighbourhoods; schools where smoking is common). They also
tend not to be succeeding according to their own or society’s terms
(for example, they have low self esteem, have impaired psychological
wellbeing, are overweight, or are poor achievers at school). The
desired image is sufficient for the novice smoker to tolerate the
aversion of the first few cigarettes, after which pharmacological
factors assume much greater importance. Again in the words of Philip
Morris, “as the force from the psychosocial symbolism subsides, the
pharmacological effect takes over to sustain the habit.”Within a
year or so of starting to smoke, children inhale the same amount of
nicotine per cigarette as adults, experience craving for cigarettes
when they cannot smoke, make attempts to quit, and report
experiencing the whole range of nicotine withdrawal symptoms.
Physical and psychological effects of nicotine
Absorption of cigarette smoke from the lung is rapid and complete,
producing with each inhalation a high concentration arterial bolus
of nicotine that reaches the brain within 10-16 seconds, faster than
by intravenous injection. Nicotine has a distributional half life of
15-20 minutes and a terminal half life in blood of two hours.
Smokers therefore experience a pattern
of repetitive and transient high blood nicotine concentrations from
each cigarette, with regular hourly cigarettes needed to maintain
raised concentrations, and overnight blood levels dropping to close
to those of non-smokers.
Nicotine has pervasive
effects on brain neurochemistry. It activates nicotinic
acetylcholine receptors (nAchRs), which are widely distributed in
the brain, and induces the release of dopamine in the nucleus
accumbens. This effect is the same as that produced by other drugs
of misuse (such as amphetamines and cocaine) and is thought to be a
critical feature of brain addiction mechanisms. Nicotine is a
psychomotor stimulant, and in new users it speeds simple reaction
time and improves performance on tasks of sustained attention.
However, tolerance to many of these effects soon develops, and
chronic users probably do not continue to obtain absolute
improvements in performance, cognitive processing, or mood. Smokers
typically report that cigarettes calm them down when they are
stressed and help them to concentrate and work more effectively, but
little evidence exists that nicotine provides effective self
medication for adverse mood states or for coping with stress.
A plausible explanation for why smokers perceive cigarettes to be
calming may come from a consideration of the effects of nicotine
withdrawal. Smokers start to experience impairment of mood and
performance within hours of their last cigarette, and certainly
overnight. These effects are completely alleviated by smoking a
cigarette. Smokers go through this process thousands of times over
the course of their smoking career, and this may lead them to
identify cigarettes as effective self medication, even if the effect
is the negative one of withdrawal relief rather than any absolute
improvement.
Regulation of nicotine intake
Smokers show a strong tendency to regulate their nicotine intakes
from cigarettes within quite narrow limits. They avoid intakes that
are either too low (provoking withdrawal) or too high (leading to
unpleasant effects of nicotine overdose). Within individuals,
nicotine preferences emerge early in the smoking career and seem to
be stable over time. The phenomenon of nicotine titration is
responsible for the failure of intakes to decline after switching to
cigarettes with low tar and nicotine yields. Compensatory puffing
and inhalation, operating at a subconscious level, ensure that
nicotine intakes are maintained. As nicotine and tar delivery in
smoke are closely coupled, compensatory smoking likewise maintains
tar intake and defeats any potential health gain from lower tar
cigarettes. Similar compensatory behaviour occurs after cutting down
on the number of cigarettes smoked each day; hence this popular
strategy fails to deliver any meaningful health benefits.
Socioeconomic status and
nicotine addiction
An emerging
phenomenon of the utmost significance over the past two decades has
been the increasing association of continued smoking with markers of
social disadvantage. Among affluent men and women in the United
Kingdom, the proportion of ever smokers who have quit has more than
doubled since the early 1970s, from about 25% to nearly 60%, whereas
in the poorest groups the proportion has remained at around 10%.
Part of the explanation for this phenomenon may be found in the
growing evidence that poorer smokers tend to have higher levels of
nicotine intake and are substantially more dependent on nicotine. It
is evident that future progress in reducing smoking is increasingly
going to have to tackle the problems posed by poverty.
Smoking as a chronic disease
Cigarette dependence is a chronic relapsing condition that for many
users entails a struggle to achieve long term abstinence that
extends over years or decades. Successful interventions need to
tackle the interacting constellation of factors—personal, family,
socioeconomic, and pharmacological—that sustain use and can act as
major barriers to cessation.
Referencias
Encuesta Mundial del Tabaquismo en Jóvenes (EMTJ)
(28/Jul/2003) en español
(407.86k)
La Asamblea Mundial de la Salud contemplará la adopción de un
tratado mundial para el control del tabaco
(15/May/2003) en español
(3.54k)
Prevalence of and risk factors for cigarette smoking among
adolescents in South America: a systematic literature review
(15/May/2003)
in english
(4.12k)